 Purred: Sun May 20, '07 10:25am PST |
 | |  | | I've copied and pasted from the Merck Veterinary Manual for you. Pretty much, the general consensus is that it's a predisposed genetic condition normally caused by a hypersensitivty reaction. For cats, its normally environmental, insect or dietary.
Does the food you're feeding right now have a few protein sources? You may want to try one that just has a couple ingredients. California Natural for instance has very limited ingredient food.
One of the things recommended by Mercks is a dietary elimination trial. Pretty much you just try one protein and carb combo and see if that's the problem. If it is, then try a different one.
Here's the link for California Natural foods:
http://www.naturapet.com/brands/california-natural.asp
And here's the link to Mercks
http://www.merckvetmanual.com/mvm/index.jsp?cfile=htm/bc/724 00.htm&word=Eosinophilic%2cUlcer
The etiology of this group of diseases that affects cats, dogs, and horses has focused on an underlying hypersensitivity reaction. This is particularly true in cats and horses. Insect, environmental, and dietary hypersensitivities have been documented in cats, while insect hypersensitivity has been seen in some equine cases and in a smaller number of canine cases. Genetic predisposition and bacterial infections have also been seen in cats. In all species, idiopathic cases exist. Eosinophilic Ulcer: This well-circumscribed, erythematous, ulcerative lesion, usually not painful or pruritic, is usually found on the upper lip. Although reported to occur, progression to squamous cell carcinoma is extremely rare. Histology shows an ulcerative dermatitis, with a cellular infiltrate of neutrophils, plasma cells, and mononuclear cells predominating. Mild to moderate fibroplasia is common. Tissue or peripheral eosinophilia is uncommon. Treatment:
In cats, hypersensitivity disorders (allergy to fleas, food, or inhalants) should be investigated by allergy testing (intradermal or in vitro) and dietary elimination trials. Hyposensitization, insect control, and dietary management should be instituted when appropriate. Antibiotic therapy (amoxicillin-clavulanate, cefadroxil, or fluoroquinolones) should always be tried empirically, especially in refractory cases. If no underlying cause can be determined and the condition is refractory, corticosteroids, such as methylprednisolone acetate (4 mg/kg, IM, once every 2 wk for 2-3 injections), oral prednisolone (2-4 mg/kg/day), or oral triamcinolone (0.8 mg/kg/day), can be tried. Oral corticosteroids should be tapered to alternate days (or to every third day in the case of triamcinolone), and dosages reduced when used for longterm management. Long-acting injectable methylprednisolone acetate should not be used more often than every 6-12 wk due to the potential for inducing hyperadrenocorticism. |  |  | |  |
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